Do I Have a Poligrip Lawsuit Claim?

New research has led to the filing of hundreds of lawsuits by denture cream users who claim that products such as Fixodent and Poligrip caused them to develop a severe neurological disorder known as neuropathy. In 2008, researchers at the University of Texas published a landmark study in the medical journal Neurology that suggested a link between the zinc in denture cream, used to aid adhesion and eliminate odors, and nerve damage. Since then litigation has ensued nationwide, with plaintiffs alleging that manufacturers such as GlaxoSmithKline (Poligrip) and Procter & Gamble (Fixodent) failed to warn consumers about the dangers of their products.

Although zinc is a required mineral in small doses, ingesting too much can lead to low copper levels in the body, which in turn can cause nerve damage. Neuropathy may initially cause symptoms such as poor balance and loss of sensation in the extremities and in severe cases, could lead to paralysis. Many of the injured plaintiffs bringing suit have been forced to stop working and require a cane, walker, or wheelchair to get around and some may require lifelong care. The physical, emotional, and financial impact of such injuries are immense, and are why many plaintiffs are seeking compensation from denture cream manufacturers such as GSK and P&G.

The allegation that these drugmakers failed to warn the public about the dangers of denture cream zinc, as well as the expectation of compensation by injured consumers, are supported by product liability laws. These laws provide protection for individuals injured by products by holding the entities that make those products available, including manufacturers, distributors, suppliers, and retailers, responsible for injuries they cause. Denture cream plaintiffs are specifically invoking “marketing defect” (or “failure to warn”) product liability laws.

When the University of Texas study was first published, GSK and P&G maintained that their products were safe when used in recommended amounts. But with lawsuits beginning to mount, GSK removed the zinc from its Poligrip products. P&G, arguing that Fixodent contained only half the zinc as Polident, continues to manufacture denture cream containing zinc. It remains to be seen how either company’s stance affects the pending litigation against them. Hard to overlook, however, is the fact that neither manufacturer even listed zinc as a denture cream ingredient until the University of Texas findings were published.

Which begs the questions: What did the manufacturers know and when did they know it? Although denture cream is regulated by the FDA, its qualification as a “Class I” medical device means it is considered low risk, and makers are not required to list ingredients on their labels. Nonetheless, all manufacturers owe consumers a duty to protect them from harm, and based on the availability of scientific literature linking zinc to nerve damage, it would seem that some denture cream manufacturers violated this duty.

For decades there has been a well-established link between excessive zinc levels and depleted copper levels. In 1992, a study was published that tied copper deficiency to neurological disorder. And earlier this decade, two studies were published that demonstrated zinc’s potential neurotoxicity and ability to cause nerve damage. Thus, even though studies such as the one from the University of Texas only recently established a relationship between the zinc in denture cream and neuropathy, the connection between zinc toxicity and nerve damage has been known for quite some time. A prominent Vanderbilt University neurologist who published findings that support those of UT researchers stated that the link between excess zinc and neurological disorders are “…nothing new. If you researched the field, you would find out.”

Which means that GlaxoSmithKline and Procter & Gamble either did not research or else knowingly withheld the results of their pharmacological sleuthing. If the attorneys representing injured denture cream users can prove the latter, this would certainly entitle their plaintiffs to compensation. But it also seems reasonable that international corporations that regularly spend millions of dollars on research and development would uncover the catastrophic side effects of the products they sell. If the courts determine this, it could also mean financial reparations for denture cream injury victims.

If you used a denture cream such as Fixodent or Poligrip and developed hyperzincemia (zinc poisoning), hypocupremia (low copper levels) or neuropathy, then you too may have cause to take legal action against GSK, P&G, or another manufacturer. To discuss the validity of your Fixodent or Poligrip lawsuit, contact the Rottenstein Law Group today. Our product liability attorneys have been helping injured consumers take on large corporations for more than 25 years. We know what it takes to hold negligent companies liable for their actions and will treat you with the compassion you deserve during this trying time.

Have you experienced complications from using Poligrip as your denture cream? Learn more about Poligrip lawsuits and see what you can do.

Examination Of A Sluggish Person

Sluggishness is often cited by those in neurology as a sickness. This makes it vital to study about the composition of sluggishness.

The old persons are often the prey. And also it is prominent that the old ones only frequently go to the physicians. In order to heal them, the review of the slowness or dizziness is the key.

Tardiness has many bases to it. Drinking alcohols continuously harm the head and result in neurological dismay.

Sluggishness cannot be healed in a short period of time. The above said reasons are be taken into account when dealing with the cure for the affected persons.

In the early stages of healing, it is important to study the past life of the suffering people.

It is due the facts that will ensure the physician to initiate nice healing. The slownesss can be subdivided in to four kinds depending on the patients moaning.

They are termed as vertigo, Presyncope, imbalance, dizziness and many other causes which the people talk about the hanging sort of feeling to the physician.

Analysis of these types one by one:

Vertigo: The affected person can be diagnosed easily by the physician with his behavior. Because such persons feel very high and they speak to others as if they are the ones who know everything.

Pre-syncope: This is a kind depending on the weakness. This makes the infected people to stand in a high speed and makes to sense the butterflies in front of the face.

Disequilibrium: It is linked to the people’s balancing strength. Such people may stumble to walk. If the physician identifies such people then he will conclude that the affected person has the imbalance trouble.

Suppose a person with imbalance problem and if he is able to sense when someone is touching his body then the problem is curable. And the imbalance is very much true if the person really struggles during night or when bathing.

The roots for this kind of trouble are because of the eye sight trouble which becomes entirely dark.

The aged ones often are prone to multiple categories of sluggishness. It’s the physician who finds all the categories that the person is affected with.

Importantly slowness is divided in to two sorts and they are reliable or harass. And any other signals such as listening problems, illness and such things support estimation of the sickness.

Review of the primary categories of slowness:

Reviewing the vertigo: Inside the ear if the signal range result in any damage to its equilibrium then it end in vertigo.

There are two kinds: Minor and Inner. The minor kind creates damage to the deep of the ear. The reception inside the ear called as vestibulocochlear nerve is majorly damaged.

In the inner kind the trouble is based on neurological disorder. Unlike the damage to the ear, this damages the brain even though there are no big indications. It should be properly treated and should not be avoided.

Cure at the right time will seriously prevent the neurological problems. The primary indication of the minor problem is the lack of self changes to the eye’s movement.

The eye’s rotation and changes to the movements to the eyes are directly controlled by the vestibular part. Listening problem is another indication for the minor kind.

But in the vital sort of vertigo there are no such listening troubles. Harassments are often in secondary sort of vertigo. Such sorts of harassments are minor.

Posture in vertigo: It is termed so because this focus on the posture of the suffering person.

Take for example if the suffering person is trying to remove a book from the shelf which is located in unreachable heights or straining and tying the shoe lace or rolling over the bed. Under such circumstances the vertigo begins immediately.

Such kind of vertigo is the result of the alterations to the locations of calcium crystals available inside the ear and they are called as otoliths. Sluggishness is assessed in this way.

If you’ve been searching for a natural healing for vertigo, check out our simple, step by step program that not only briefs vertigo effects but also includes simple exercises that heal dizziness permanently.

Aspirin Jabs More Effective For Migraine Headaches

Migraine sufferers can take a jab (injection)made from liquid aspirin to relieve their severe headache. The news comes from the research conducted by the researchers from The National Hospital for Neurology and Neurosurgery in London and the University of California. It was published in the medical journal Neurology.

The previous studies have reported intravenous or IV aspirin to be effective for treating acute migraine. The present study wanted to examine if IV aspirin could also be effective against chronic daily headache, particularly in people who had severe headaches because of the overuse of headache relief medicines.
The patients of migraine often withdraw from medications such as paracetamol, opioids or triptans used to treat migraine. The same medicines cannot be used to treat the withdrawal headache in people with migraine.

The research involved identification of all people who had received IV aspirin for severe headache between the September 2001 and May 2006. They used patient diaries and other medical records to assess the characteristics of these people and the effect of IV aspirin on their pain.

The final sample consisted of 168 people who had received IV aspirin. The level of pain was assessed on scale of 0 to 10 with 0 representing no pain and 10 higher level of pain. It was measured three hours before giving a dose of aspirin or chlorpromazine or aspirin. The participants were given 1g doses of aspirin intravenously for five times in a day.

Around 25 percent of the people reported a decline in pain to a drop of three and about 40 percent of the people had modest fall in pain to about two points. The final conclusion from the research was that IV is a safe and effective treatment for managing severe headaches in people admitted to hospital.

The study gives an indication that the treatment can be safe in people with severe daily headache but there are a number of points to be considered about this study. The study used patient diaries that were not designed to assess the effects of IV aspirin. And about half the patients did not maintain a diary so again there might have some differences in the experiences of such people.

The most important point is this study concentrated mainly on the headache caused due to overuse of pain medication and therefore cannot be generalized to a general population who experience headache and migraine. The study needs to be conducted on a larger population to test the effectiveness of IV aspirin before it can actually be used in treatments.

Migraine affects a large number of people and the pain is really severe. The intravenous aspirin can be very effective in treating severe headache as a result of overuse of medication. It will also be of fewer burdens on a patients pocket in terms of price.

Dr. Anita Brown is a New York based Physician. She actively contributes articles to on a wide range of topics and interests. HealthEnclave is the one stop for Health, Fitness and Wellbeing. Visit for the latest in Health and Wellbeing.

ICD 9: Updated CVA Diagnosis Guidelines

With the ICD 9 codes 2011 going into effect on October 1, 2010, as a neurology coder, you too can expect some changes likely to affect your practice. Here are some guidelines that will stand you in good stead:

Check your terminology

Patients and practitioners sometimes use the terms ‘stroke’ and ‘CVA’ interchangeably to refer to a cerebral infarction. The terms ‘stroke’, ‘CVA and ‘cerebral infarction NOS’ each fall under diagnosis 434.91. The updated guidelines add, ‘additional codes should be assigned for any neurological deficits with the acute CVA, irrespective of whether or not the neurologic defect resolves before discharge’.

Do not mix late effects with neurological deficits

Diagnoses under ICD-9’s category 438 deal with late effects of cardiovascular disease. A late effect is the residual effect post the acute phase of an illness or injury has terminated. There is in fact, no time limit on when you can use a late effect code.

According to ICD 9 2010 guidelines, you needed to turn to 438.xx when indicating conditions in categories 430-437 as the cases of late effects. These late effects include neurological deficits that persist after the initial onset of conditions in categories 430-437, like speech and language deficits (438.1x), dysphagia (438.82), or vertigo (438.85).

With effect from October 1, 2010, guidelines specify to use codes in category 438 only for late effects of cerebrovascular disease, not for neurological deficits associated with an acute CVA.

Diagnosis signals disease

Guidelines under Section 1 C.18.d.3 differentiate status and history diagnosis codes. The guideline update clarifies what status codes represent. These codes indicate that a patient is a carrier of a disease, has the sequelae or residual of a past disease or condition, or has another factor influencing a person’s health status.

A status code (such as V58.61, Long-term use of anticoagulants) informs healthcare providers and insurers of the patient’s condition and might affect the course of treatment and its outcome. Using a personal history code (like V12.41, Personal history of benign neoplasm of the brain) explains a patient’s medical condition that no longer exists and is not receiving any treatment. The code also indicates that the patient has the potential for recurrence, and therefore might need constant monitoring.

Since all ICD-9-CM 2011 books will not include the updated guidelines as the books went to printers before the updated guidelines became available, you’ll stand in good stead to sign up for a medical coding guide like Supercoder!

We provide you simple, instant connection to official code descriptors & guidelines and other tools for 2010 CPT code, HCPCS lookup that help coders and billers to excel in the work they do every day.

Acupuncture Treatment: Acupuncture for Muscular-skeletal & neurological

Acupuncture is a very old and popular technique from traditional Chinese medicines. It is very ancient and since then practiced to cure various diseases in patients. This technique due to its popularity has spread all over the world. The western medical system has worked on its principles and evolved with new techniques in acupuncture theses techniques are improved version of basic acupuncture. Acupuncture has proved itself helpful for circulatory, respiratory, muscular, neurological, and digestive disorders. Besides that, it helps to give you a beautiful skin through techniques involved in cosmetic acupuncture, and auricular acupuncture is helpful for all body systems.

The original acupuncture treatment uses plastic or glass needles which are inserted in certain points in the body. These points are termed as acupuncture points. They are located on the energy channels in the body. Each acupuncture point is associated with a body part so their pinching causes stimulation ion that organ. The energy channels are called as meridians which carry energy all through the body. Any disorder in body is caused due to the blockage in pathways of energy and acupuncture always works to open those blocked paths. Western practitioners have evolved different methods to simulate organs, they uses laser, electric impulses, magnetic radiations so that pain involved in acupuncture can be avoided.

Among the various neurological disorders on which acupuncture works comes the headaches. It is effective in nay type of headache like migraine headaches, tension and cluster headaches. It causes an immediate effect on headaches and prevents their frequent occurrence. Some neurological disorders include stagnation in nerve functioning also called nerve degenerating disorder. When needles are inserted in nerves, it causes stimulation at the point and thus regaining their activity. Sometimes electric stimulation is also used for nerve stimulation. Besides these, it is helpful in stroke rehabilitation, damage due to stroke like hardening of arteries, hypertension and head injuries and treats kinds of tumors. This is also used during brain surgery and supports treatment of Parkinson’s disease, cerebral palsy, epilepsy, nerve deafness

Acupuncture treatment is famous for its effect in pain and stiffness in muscles. At times, sue to stress and fatigue muscles get contracted and their movement is restricted. With acupuncture on muscles related points, muscle movement is restarted. It increases blood flow and oxygen to muscle tissues and thus providing them necessary nutrition and energy to work actively. Diseases like myofascial muscle syndrome which causes acute pain in neck, shoulders, elbows and knee have great effect of acupuncture. Along with these, tendonitis, strains inflammation and sprains in muscles are also removed with acupuncture. In bone fracture chronic pain can be avoid with acupuncture if bones are damaged from sprain and tear in tendons and ligaments.

Thus this acupuncture treatment is effective in various muscular, skeleton and neurological disorders. This therapy works for those also who are tired of taking drugs but do not see any results. Also for those who do not want to take harmful medicines, this method is beneficial. But you have to choose a licensed practitioner to avoid any infection and other harmful effects from this treatment. This is very safe and natural technique with no pain and no harm.


George Jhon is an Author for Backonyourfeet, An Acupuncture Sydney clinic where our highly skilled experts may guide you about Fundamentals of Acupuncture Treatment to Improve Your Life. For more information, visit


West Nile Neurological Disease – Part 3 – Focus on Faith

In 2007, West Nile Neurological Disease paralyzed my husband, Rick, in three limbs, and caused multiple complications, including encephalitis. The virus nearly stole him from me, but mercy sent us instead into a long exile at a rehab centre several hours from home. All along what has now become a multi-year journey, we have clung tightly to God.

We had practiced for this: my husband has been a Christian clergyman for over thirty years. I’ve been a Christ-follower since childhood. Faith is central to our lives. Part of that faith is our belief that though the world is rife with disaster and difficulty–to which we are not immune–we have a living, loving God who walks beside us, holds us up when we can’t stand, and pays attention to our prayers. During our journey down the Nile, we fed our faith in many ways. Here are a few:

Likely the most spiritually nourishing practice became our reading of scripture. Daily, as I sat by my husband’s bed, I read to him aloud from the Bible. He often told me how those ancient words from which he had preached for three decades, were the only thing that took his mind off his paralysis and constant, traumatizing pain.

We prayed together often–holding hands. Because Rick’s encephalitis and pain made this familiar habit difficult, I did most of the talking aloud to God. He told me he found that extremely healing.

We made time for regular enjoyment of nature. The rehab centre had a solarium to which I pushed Rick often, simply to sit and enjoy the changing seasons outside the tall windows. Chickadees, sparrows, even rabbits, entertained us early in the mornings. As he improved, I was able to wheel his chair outside where we “rolked” (rolled and walked, a term coined by another patient who had tragically lost her legs) down to a path that followed outline of a small lake.

Even when we didn’t feel like it on many Sundays, and even though the services weren’t the kind we were accustomed to, we took in the weekly chapel service the facility offered. Surrounded by others in wheelchairs, we experienced the “fellowship of the similarly afflicted”–a bond with other disabled people that endures to this day, even though Rick has graduated to a walker.

We made time for music that boosted us spiritually. Not always gospel songs, but especially gospel songs. Though the old hymns of the church sung as commonly these days, they kept creeping out through the fabric of our joint memory to encourage us. I often sat at the piano in the common area and played them by memory. Other patients and family members joined us sometimes. God, we know, did too, regularly.

I sang one of those hymns almost every morning. It emerged without waiting for an invitation, almost immediately after waking. The archaic words seemed both relevant and crucial for me: Oh, for a faith that will not shrink, though pressed by every foe; that will not tremble on the brink of any earthly woe. Lord, give me such a faith as this…!

I also spent hours reading–after getting into bed and before rising. From other people of faith, who had written on the subjects of pain, grief, and loss, I gained new perspectives, which I often shared with Rick. Those words have served us well all along the Nile.

If pirates have boarded your boat, I encourage you to board an even larger one – the stable boat of spiritual direction, comfort, and provision. Keep (or develop) an active faith in God. It will be your most effective weapon in battle.

You’ve just read article three in the E-Zine series: West Nile Neurological Disease–Fighting Life’s Pirates.Read the complete story of our West Nile journey in West Nile Diary, One Couple’s Triumph Over a Deadly Disease available on Amazon.

P.S. Wear repellent.

copyright 2010, by Kathleen Gibson. If you copy, copy right, and for non-profit use only. Please include author credit, and a link to my website, below.

Kathleen Gibson is a Canadian author and newspaper columnist whose work has been published in global print and online media. Through articles, interviews and her book, West Nile Diary–One Couple’s Triumph Over a Deadly Disease, she and her husband, Rick, have raised the level of West Nile Disease awareness across North America. Together, they point others to the beautiful strength that comes from cultivating a solid faith in God. Kathleen’s latest book is Practice by Practice, The Art of Everyday Faith. More info at

Boundaries of Self – Representational Self and the Neurological Representation of an Individual

Logic dictates that entity A cannot be entity B, and when we come to valuate that do the representations we have from ourselves truly represent ourselves, in relativity with this simple logic, the answer must be “No”. The entity we are perceiving in our consciousness is combined of units of information, and is a virtual entity, similarly with our friends and foes we perceive in our consciousness even when they are not around. And these representations lack the neurological detail, everything that we know as the physical basis of our existence, every known form of self-awareness, and are immaterial in the environment. And as our sense of the environment is representational, when the external source of an individual we know is not at present, the representation of the individual we have in our consciousness does not have a mind of its own, but is the exact then active image we have from him/her, each of them produced in relativity with our own internal complexity and the dynamics of the sub-consciousness.

It must now also be acknowledged that our individuality, not the soul, is defined also in neurological, cellural and in genetic level, i.e. that the combination of atoms in molecular level is what makes us human individuals, having the 1-2% difference in genetic level, making us unique in physical design. Yet, in the experiments done through cloning also indicate through deductions that, the distinctive observer/soul cannot be replicated however identical the genome that produces the chain reaction of combining the body were between mammals, not to mention that the combination of genes that is able to construct the individual self of us does not emerge anywhere in the entire cosmos and beyond during our existence, transcending us to those spatial co-ordinates. If the observer/soul was somehow linked between the two cloned Dolly sheep, it would have required some Cartesian receiver-transceiver device, enabling them to send and receive neural impulses from each of other in order to build collective self-awareness, but their genome had no such blueprints for it to be formed. Ego, or Das Ich (The I, or I itself) as Freud would have expressed or soul is therefore not something that can be replicated nor can it be divided to multiple bodies, forming a collective consciousness with an individual (one) ego.

In order for information to be in consciousness, it needs neurological support system that can contain information. This is the same with innate information systems in neurological level, for without qualifying and distinctive properties, the neurons and specialized receptors in various parts of the brains and for example neurons that are responsible and that enable motor tasks could not exist. Again, memory cells and for example synapses require distinctive molecular combination of atoms in order to exist and therefore units of information these memory cells and synapses contain must also have a unique molecular combination per unit of information, since otherwise contained units information should be considered as some magical meta-impulses that are contained in memory cells, identical in molecular combination, although containing different units of information. But on the other hand, energy in its form of information in neurological cognitive systems is yet without any recognizable types, i.e. that for example this waveform means “a peanut”, and this “Plato”, and the combination of these is waveforms is “Plato in a peanut” and even more, the problem of indicating which memory cell contains the searched unit of information is limited by the lack of technology of being able to scan neurons in the level of molecular combination. The distribution of information that are combined to a representation from different areas and lobes of the brains cannot be traced in the level of finding specific units of information. But non-the-less, the way our retina deals with the individual photons is that they are translated to neural impulses and are first sent to individual neurons in the visual cortex until sent to various perception-relative regions of brains to form the big picture (V3-V5), during the content generative cycle before representations emerge to consciousness, with parallel categorical types of associated information (from dimensions of information such as color, taste, semantics, emotions, sounds, ect. from other neurological systems). Because of the technological limitations, the only way to approach this matter at hand is logic. The first logical deduction is that the brains form representations from combined units of information, with the content generated by the micro-level neurocomputation for the individual’s consciousness. The second logical deduction is that because different areas of the brains perform different tasks with relatively different types of information, the information that consciousness receives as the complete representation of the environment is composed of units of information individual areas of the brains have processed. The third logical deduction is that brains are in constant constructive state in relativity with the information translated by senses of the environmental conditions and in relativity with the information already contained by the memory systems. And the fourth and most obvious logical deduction is therefore that the categorical increasing complexity defines the units of information that is to be used by the constructive and re-constructive behavior of our brains while building representations. The same deductions of this re-constructive behavior of our brains to build representations from units of information in diverse memory systems were made by F. C. Bartlett in as early as 1932:

“The first notion to get rid of is that memory is primarily or literally reduplicative, or reproductive. In a world of constantly changing environment, literal recall is extraordinarily unimportant. If we consider evidence rather than supposition, memory appears to be far more decisively an affair of construction rather than one of mere reproduction”

Now, in neurological representation of an individual, i.e. the molecular combination of an individual containing all the units of information, including the various information systems in the levels of individual neurons, memory cells, sysnapses, ect. and the systems and representations they produce in combination and as sums of their parts, the innate neurological composition produces the frame of value-relative constancies, with the individual genetic differences. The increasing complexity (for example growth of dendritic spines for an anatomical substrate for memory storage and synaptic transmission) after the birth in relations to the information of the environment and coping with the reality can for example exceed in such categories as motor tasks, emotional responses, learning to recognize objects by the smell value-relatively combined to them, ect. Virtual information such as representations of ethical behavior virtually extends the innate behavioral patterns and produce virtually extended value-relative behavior, increasing the complexity of the neurological representation of the self. The existing virtual information about the atomic combination of the environment, i.e. the atomic quantum reality to make the distinctive difference between the information containing and not-containing environment, chemical reactions and the laws of physics virtually extends the innate capacity on understanding the environment. Religions extend the innate nature virtually to a spiritual dimension. The information society systems virtually extend the innate group behavior. I mention these examples because the neurological representation of an individual is extended by virtual information, that is, an individual is also the information he/she contains, adding a third dimension which is beyond the Cartesian dualism. The increasing complexity in the category of etiquette increases the neurological representation of an individual in relations with the behavior in accordance with etiquette. As the characteristics are again considered as units that are universal, the active combination of them defines that area of the neurological representation of an individual. The more one characteristic increases in complexity, the more units of information exists in such characteristic in the neurological representation of an individual. Thus, the more one virtually extends one’s innate nature in the dimension of smart business transactions, the more virtually extended the innate nature the neurological representation of an individual becomes.

Now, as it isn’t the representational self that moves in the environment, but the neurological representation of the self, it is healthy to make a clear difference between these two distinctive levels of being. The representational self is a fantasane entity, and the neurological self is the authentic manifestation of an individual. The way we move in the atomic reality is in relativity with the active neurological combination in our micro-level neurocomputation, and its dynamics are in constant state spatial shifts. These shifts and the active combination of them define exactly where we are in the mental space, and the place our physical being manifests is defined by the spatial co-ordinates we exist in in the space-time continuum of the cosmos. In both cases, where our neurological and atomic existence is located, it is the exact location we exist in.

Henry M. Piironen was born in 1982 in Jyvaskyla, Finland, and has studied complex adaptive systems, systems theory, information theory, memetics, cosmology, the human brain neuroanatomy, theories of consciousness, self-organization, co-adaptation, emergence and religions. He experienced scientific enlightenment in 2007, leading to the creation of the model of information reality, which is addressed in his first book, “The Art of Perception: An Introduction to Information Reality” (2008). His current studies are on the information-driven continuums of superorganisms and how information extends the mind. For more discoveries of the information driven world, visit his official website at:

5 Neurological Complications Of Lyme Disease To Be Aware Of

Lyme disease, left untreated, may cause a number of debilitating neurological conditions that attack the body and mind.

People get Lyme disease from the bites of infected ticks. The early signs of Lyme disease are inclusive of a characteristic rash which appears around the part of the body where the bite occurred, fever, chills, headaches, muscle and joint pains, and swollen lymph nodes. Lyme disease shares symptoms with more common illnesses and as a result people that have contracted it end up using inappropriate treatments or miss getting treatment entirely. Unfortunately, when left untreated, Lyme disease can lead to severe complications. In the second stage of Lyme disease, neurological complications like numbness, severe headaches, and visual disturbances may occur. Lyme disease is associated with the following neurological complications:


A very painful radiculitis is among the first neurological complications that are experienced by people with Lyme disease. A radiating pain along the dermatome of a nerve often characterizes radiculitis. This condition generally occurs within the first few weeks to months of the infection. Patients usually experience sensory, motor and mixed symptoms and conditions like weakness and sudden reflex and sensory changes. Unfortunately, these symptoms are sometimes mistaken as indicators of nerve-impingement.

Cranial neuropathies, or head symtoms

As well as the patient’s cranial nerve, cranial neuropathies can affect the nerves involved in sensory processing, in other words sight, sound, smell, taste and touch. As Lyme disease progresses the multiple cranial nerves of can all be affected at the same time in someone infected with the disease. An estimated 50%-70% of total patients that suffer from neurological symptoms experience this complication.

Intracranial hypertension

A rare neurological complication of Lyme disease is intracranial hypertension. It is more commonly experienced by children and adolescents. Headaches and papilledema are common side effects associated with intracranial hypertension. Papilledema, the swelling of a patient’s optic disc, develops over a span of a few hours to a few weeks. In some cases, abnormalities in Cerebrospinal Fluid may also occur.

Inflammation of the brain and spinal cord

Sometimes resembling ischemic patterns, encephalomyelitis usually includes brainstem abnormalities. It can be medically proofed that the parenchyma is involved. Cerebellar syndromes, motion disorders, hemiparesis, and spastic paraparesis are all indicators of this complication. This complication is more common in European nations than in North America.


Encephalopathy is one of the most common complications that occurs in the later stages of Lyme disease. Those with this problem experience minor or even major cognitive changes and polyradiculoneuropathy. Encephalopathy as well as other late stage infections may be accompanied by severe fatigue, sleeping problems, extreme irritability, mood swings, photophobia, difficulty finding words, and problems in writing or speaking. There have been reports of sensory issues as well. The severity of these symptoms may differ and are considered to be somewhat inconsistent.

Early identification of the symptoms of Lyme disease and administration of immediate treatment are the best ways to prevent it from developing into worse neurological complications.

To learn more about various URL, go to

10 Lyme Disease’s Alarming Neurological Symptoms That One Should Look Out For

Some neurological symptoms of Lyme disease which you should look out for are numbness, abnormal feelings in the limbs, Bell’s palsy, meningitis, vision problems, difficulty in concentrating, memory loss, encephalopathy, sleep disturbances, and also cognitive impairment.

Lyme disease is an infection that results from the bite of an infected tick. The illness is caused by bacterium borrelia burgdorferi. People who live in grassy and wooded locations should be cautious of ticks, which are usually common in those areas, and take every possible precaution when going outside. Symptoms of Lyme disease include a rash in the bite area which spreads over time and many flu-like symptoms, such as fever, body pains, fatigue and chills. Besides the usual symptoms that manifest in the early stages of Lyme’s disease, there are certain neurological symptoms that one has to look out for which show up a few weeks or even many years later.


Numbness is an indication of diseased or damaged nerves. Several people describe the feeling as something similar to pins and needles on their skin, and people suffering from Lyme disease will often feel a prickling or burning pain in the affected area. Numbness causes patients to experience a loss of sensation unlike paralysis where patients are unable to move.

Abnormal feelings in the limbs

Patients often say that their arms and legs will feel weak. Tingling sensations in the extremities, bruising, burning and swelling may be experienced.

Bell’s palsy

Bell’s palsy is another symptom of having a Lyme diseases. Bell’s palsy is characterized by a sudden paralysis of the facial muscles caused by complications in the patient’s facial nerves. There is often a partial or whole paralysis of the face of the patients.


Meningitis is one serious complication of Lyme disease. This condition can lead to a very rapid death as the membranes around the brain and spinal cord swell. This condition is referred as meningitis and patients can experience stiff necks and headaches which are not cured by typical over-the-counter medicines. In addition, they experience a heightened sensitivity to light.

Problems with vision

Patients may notice problems with their eyesight as well. Vision troubles may arise ranging from trouble seeing at night to total blindness or impaired vision.

Problems in terms of concentration

Lyme disease is a physical condition that is known to affect a patient’s concentration. These patients may find it difficult to concentrate on a single task at a time.

Memory loss

Memory problems are another symptom of Lyme disease. They can have difficulty recalling details and may feel disoriented when trying to remember something.


Lyme encephalopathy may result when Lyme disease is left untreated in its early stages. Some symptoms of encephalopathy are dramatic mood swings, depression and a tingling sensation in the limbs.

Disturbed sleep

The sleeping habits are changed in the people who are infected with Lyme disease. These people may suffer from apnea, insomnia and other sleep disorders and disturbances.

Cognitive impairment

Cognitive impairment can occur when a person has a Lyme disease. Thinking and making decisions may be difficult for the patients. Poor concentration and memory loss are byproducts of cognitive impairment.

It is important to be aware of the different symptoms of Lyme disease so proper treatment can be administered before the disease worsens. It is said that prevention is better than cure so it is always good to take proper precautions to avoid the illness.

To get additional information on Lyme disease symptoms, you can go to

West Nile Neurological Disease – Part 1 – Our Battle Begins

Are you intimate with one or more of life’s multitudinous pirates? Catastrophe. Illness. Accident. Sudden disability. Financial ruin. Severed relationships.

They specialize in surprise. They often attack when the little boats of our lives are in full sail, when the day is fine and the sky clear. When we least expect trouble. Suddenly, without warning, the life we knew is no more. Gone. Flipped upside down as easily as a donut in hot oil.

We’ve lived with a few pirates, my husband and I. The last batch entered our lives quietly a few years ago. It happened in an instant. We didn’t even notice their invasion at first. They arrived in the form of an unseen, unfelt, common summer occurrence. My husband got bit by a mosquito.

The mosquito, we know now, imported a virus onto the little boat of our lives. In the space of a few short days, my husband, Rick, then an active fifty-four year old clergyman, became cognitively disoriented and paralyzed in both legs and his left arm.

Doctors diagnosed him with a severe case of West Nile Neurological Disease (also called West Nile Neuroinvasive Disease). How ironic that the pirate that led the attack was no bigger than his baby fingernail.

After eleven days in ICU, and another almost three weeks on a regular ward, only moments of which he actually remembers, doctors transferred Rick to a rehab center two hours from home.

On a blue and gold September afternoon, I locked our front door behind me and followed the ambulance down the silver stream of highway, past ripening fields of wheat and barley, past herds of grazing cattle to a rehab centre in a nearby city. We stayed there for the next five months, my husband in a double ward room, and I in a rented hostel room right in the centre.

Though he’d already been in hospital for almost a month, it was in that rehab centre that we began learning about what it really means to do battle with the pirates of West Nile Neurological Disease.

We also learned something else: many of the strategies and tools we found helpful are common to all life’s difficulties, no matter the stripe of pirates-or monsters, or madmen, or beasts.

If you’re fighting pirates, be sure to read this entire article series–this is the first. If you’re not fighting pirates, read be sure to read this entire article series. Life can change with the suddenness of a summer squall. One day you’ll need the information you’ll find in one or more of them.

You’ve just read article one in the E-Zine series: West Nile Neurological Disease–Fighting Life’s Pirates.

Read the complete story of our West Nile journey in West Nile Diary, One Couple’s Triumph Over a Deadly Disease available on Amazon.

P.S. Wear repellent.

copyright 2010, by Kathleen Gibson.

If you copy, copy right, and for non-profit use only. Please include author credit, and a link to my website, below.

Kathleen Gibson is a Canadian author and newspaper columnist whose work has been published in global print and online media. Through articles, interviews and her book, West Nile Diary–One Couple’s Triumph Over a Deadly Disease, she and her husband, Rick, have raised the level of West Nile Disease awareness across North America. Together, they point others to the beautiful strength that comes from cultivating a solid faith in God. Kathleen’s latest book is Practice by Practice, The Art of Everyday Faith. More info at